Product Code: CAS 54965-24-1
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Tamoxifen Citrate  99% Assay CAS: 10540-29-1

Quick Details:

Tamoxifen Citrate, Nolvadex
CAS NO.: 54965-24-1
Assay:99%
EINECS: 259-415-2
Molecular Formula: C26H29NO.C6H8O7
Molecular weight: 563.64
MP: 140-144 ºC
Appearance: white crystalline powder.
Usage: Tamoxifen citrate ( Nolvadex ) is a non-steroidal agent that demonstrates potent antiestrogenic properties. The drug is technically an estrogen agonist/antagonist, which competitively binds to estrogen receptors in various target tissues.

COA:

 

TestAnalysis StandardResults
CharacteristicsWhite crystalline powderconforms
Assay99 ~ 101.0%99.51%
Loss on drying≤0.5%0.24%
IdentificationIR,UV conformconforms
Residue on ignition≤0.2%0.06%
Iron≤0.005%0.0016%
Heavy metals≤0.001%0.0006%
Related substancesTotal:≤1.0%0.37%
Individual:≤0.5%0.17%
E-isomer≤0.3%0.20%
Organic volatile impuritiesconformconform
ConclusionThis batch is complies with USP 30.


Description:

Nolvadex (tamoxifen citrate) is very comparable to Clomid, behaves in the same manner in all tissues, and is a mixed estrogen agonist/antagonist of the same type as Clomid. The two molecules are also very similar in structure. It is not correct that Nolvadex reduces levels of estrogen: Rather, it blocks estrogen from estrogen receptors and, in those tissues where it is an antagonist, causes the receptor to do nothing. 
The claim that Nolvadex reduces gains should not be taken too seriously. The fact is that any number of bodybuilders have made excellent gains while using Nolvadex. 
The belief that it reduces gains seems to stem from the fact that the scientific literature reports a slight reduction (individuals using anabolic steroids were not studied though) from use of Nolvadex. 

Many women have breast cancer that tests positive for estrogen receptors (ER+). This means that estrogen promotes the growth of thebreast cancer cells. Tam0xifen blocks the effects of estrogen on these cells. It is often called an "anti-estrogen."
Tamoxifen Citrateslows or stops the growth of cancer cells that are already present in the body. It helps keep the original breast cancer from coming back and helps prevent new cancer in the opposite breast. It also reduces the risk of breast cancer in women who have a high risk for this disease.
 Tamoxifen Citrate can be used to decrease the rate of bone maturation and alter the predicted adult height, which is usually lower in those with premature puberty. This medication has been shown to be affective for both boys and girls. It was observed that there was persistent retardation of longitudinal and cortical radical bone growth, which then led to come concern about the use of tam0xifen in young humans.
Tamoxifen Citrate can be used in conjunction with other medications to form a treatment regime to regulate the growth of new blood vessels.


Usages:

The goods to anti-estrogen fertility inducer, the objects in dysfunctional uterine bleeding, polycystic ovary, menstrual disorders and drug-induced amenorrhea and other gynecologic diseases; It is used as a first line defense against breast cancer. Nolvadex or Tamoxifen citrate, a non-steroidal antiestrogen for oral administration, is commonly by athletes as a post cycle therapy drug to reduce or prevent the formation of excess estrogens that can lead to oily skin, acne, fluid retention, and gynecomastia.  

Functions:

Tamoxifen is an antagonist of the estrogen receptor in breast tissue via its active metabolite, hydroxytamoxifen. In other tissues such as the endometrium, it behaves as an agonist, and thus may be characterized as a mixed agonist/antagonist. Tamoxifen is the usual endocrine (anti-estrogen) therapy forhormonereceptor-positivebreast cancer in pre-menopausal women, and is also a standard in post-menopausal women although aromatase inhibitors are also frequently used in that setting. 

Tamoxifen is metabolized into compounds that also bind to the estrogen receptor but do not activate it. Because of this competitive antagonism, tamoxifen acts like a key broken off in the lock that prevents any other key from being inserted, preventing estrogen from binding to its receptor. Hence breast cancer cell growth is blocked.

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